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Real Calm

Are you struggling with pain?

Just ONE Medical Issue May Be The Culprit!

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What underlying cause contributes to all of the following conditions?

Inflammatory pain

Osteoarthritis (OA)

Rheumatoid arthritis (RA)

Chronic low back pain

Headaches or Migraines

Chronic neck pain

Fibromyalgia

Pelvic pain syndromes

Chronic Shoulder Pain

Whiplash

Tennis Elbow

Neuropathic pain

Irritable bowel syndrome (IBS)

Shoulder impingement syndrome

Chronic fatigue syndrome

Temporomandibular joint disorder (TMD)

The Answer Is... NMDA Overdrive!

If you suffer from any of these conditions, you need to know about NMDA Overdrive.

What is NMDA Overdrive?

Inside your nervous system – located directly on the neurons themselves – there are specialized proteins called NMDA receptors (N-methyl-D-aspartate receptors). These receptors respond to glutamate – your brain’s main excitatory neurotransmitter.

medical model of a NMDA Receptor
What makes these receptors so special is that they’re responsible for regulating the flow of calcium into the neuron, which is essential for transmitting signals related to learning, memory, neuroplasticity and pain.

Under normal conditions, your NMDA receptors help your nervous system respond appropriately to painful stimuli.

But when these receptors over-activated — for example, after an injury, inflammation, or persistent stress — it drives a process called central sensitization.

  • Central sensitization is what happens when the brain and spinal cord become hypersensitive as a direct response to overstimulation.
  • Numerous studies have demonstrated that NMDA receptor activity is upregulated in chronic pain patients of various types – which essentially locks these people into a state of persistent pain. [1, 2, 3, 4]

The good news is: There's a Safe Way To Regulate Unwanted NMDA Receptors Without Unwanted Side Effects

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What happens when you are in ‘NMDA Overdrive?’

Pain signals are amplified.

Even normally non-painful sensations (like light touch) start to feel painful.

Pain persists long after any damaged tissues have healed.

How it works:

When NMDA receptors are repeatedly activated - due to ongoing pain or injury - they allow excessive calcium to flow into neurons.

This surge of calcium causes biochemical changes that actually re-wire your nervous system to stay in a "high-alert" pain mode.

The result: Chronic pain, often without any ongoing physical injury.

It’s not a coincidence that so many chronic pain conditions have overlapping symptoms like fatigue, sleep disturbances, cognitive difficulties, and sensory sensitivities

These symptoms are representative of the complex nature and far-reaching effects of central sensitization. [5, 6, 7, 8, 9, 10]

When this happens even harmless signals can feel painful.

Think of it like this:

You stub your toe once: ouch.

Your nervous system keeps replaying that stub and now even a sock feels like fire.

That is sensitization, and it’s driven by your NMDA receptors.

Pharmaceutical companies know all about the importance of NMDA receptors …

In fact, there’s an entire class of drugs designed specifically to block NMDA receptors - Ketamine is one most that people are familiar with - and these drugs are often used to:

Reduce pain hypersensitivity

Prevent the progression of acute to chronic pain

Reset the nervous system

And they are so good at doing those things they are often referred to as “Miracle Drugs” [11]

But there’s a catch …

The catch – using them, even for short periods of time can lead to:

  • Unwanted side effects (such as dizziness, disorientation, confusion, nausea, vomiting, loss of motor coordination, increased heart rate & blood pressure as well as visual and/or auditory hallucinations). [12]
  • In adults, adverse effects of NMDA antagonists are mainly central nervous system (CNS) side effects including hallucinations, lightheadedness, dizziness, fatigue, headache, out-of-body sensation, nightmares, and sensory changes.[13]
  • Physical dependence
  • Withdrawal syndrome when you stop taking them.

Again, The good news:
There are some natural substances that can inhibit NMDA receptors as well.

The non-pharmacological approach to NMDA inhibition is rapidly gaining recognition for its important role in managing both acute and chronic pain – due in large part to “the serious risks and uncertain benefits of other options.” [14] These are some of the nutrients that have been found to be most effective for shutting down NMDA Overdrive:

Magnesium

The Natural NMDA Blocker

Magnesium has a potent anti-nociceptive effect specifically because of its ability to inhibit NMDA receptors.

Magnesium binds inside the NMDA receptor and blocks calcium from flooding in.

It acts like a cork in a bottle, preventing excessive nerve firing unless there’s a strong enough signal.

This action helps prevent the development of central sensitization and can even alleviate established pain hypersensitivity.

For every additional milligram of magnesium consumed per kg of body weight, the odds of experiencing chronic pain decreased by 8%. [15]
Unfortunately, far too many people are Magnesium deficient. According to the National Health and Nutrition Examination Survey (NHANES): About ½ of all Americans take in less magnesium than is required. [14]

Backed by research:

Magnesium has been successfully used for pain management in conditions ranging from migraines, neuropathic pain and fibromyalgia to postoperative pain. [16, 17, 18, 19]

Why it matters:

When magnesium levels in the body are too low, NMDA receptors stay overactive, fueling sensitization and chronic pain. Getting a sufficient dose of daily magnesium is an easy way to dramatically reduce pain signaling in the body.

Zinc

The Modulator

Like Magnesium, Zinc blocks the action of your NMDA receptors, but it does so through a completely different binding site that’s located on one of the receptor’s subunits. [20]

This is thought to be the main mechanism explaining the anti-nociceptive properties of zinc.

Zinc binding to NMDA receptors is a “fundamental molecular event” in principal aspects of pain transmission, chronic pain development.” [21]

This is why Zinc and Magnesium and zinc work so well together – because they act synergistically to inhibit NMDA receptors rather than competing with each other.

Multiple studies have also demonstrated that zinc is effective for calming neuroinflammation and dampening neuronal excitability. [22, 23]

Unfortunately, Zinc insufficiency is one of the world’s major public health issues. Researchers estimate that about 50% of the world’s population may be suffering from zinc deficiency.[24]

Research says:

Zinc deficiency is linked with increased pain sensitivity, poor sleep quantity & quality as well as

Bonus:

Zinc also supports GABA, your brain’s main calming neurotransmitter, which helps balance the excitation from NMDA. [22]

Selenium

The Jack of All Trades

Selenium is an essential nutrient, and like other essential nutrients (essential fatty acids, essential amino acids) – that means our bodies need it in order to function optimally but we cannot produce it so it must be consumed through diet.
  • Even though we only need a little selenium, it plays a major role in important processes inside the body, especially with regard to thyroid/metabolic function and brain and nervous system where it’s heavily involved in antioxidant defenses.
  • Like Magnesium, Selenium has been shown to modulate calcium influx, reduce neuro-inflammation and alleviate neuropathic pain. [25]
Calcium entry across membranes in the body is controlled by different channels – including transient receptor potential (TRP) channels. Some of these of which can be activated by oxidative stress and have a role in the induction of peripheral pain. The results of recent studies indicate the modulator roles of selenium in peripheral pain through inhibition of TRP channels [25] Which is why even Low-dose selenium has demonstrated the ability to reduce pain [19,20]

Selenium also has neuroprotective effects through modulation of excessive ROS production, inflammation, and Ca2+ overload (sensitization) in several diseases including”

  • Inflammatory pain
  • Hypersensitivity
  • Allodynia
  • Diabetic neuropathic pain
  • Nociceptive pain. [25]

Signs of SELENIUM DEFICIENCY

Muscle pain

Weakness

Lethargy/fatigue

Weakened immune system - Frequent colds/infections - Autoimmune disease

Hypothyroidism

Hair Loss/ Changes to hair quality/texture [26]

Selenium has another way of improving the lives of people suffering with chronic pain – improving their sleep quality and quantity.

  • Multiple studies have demonstrated that optimum sleep duration and Se intake are positively correlated [27]
  • Research showed that people who consumed large amounts of Se experience the best sleep. [28]

Vitamin B6

The Alchemist

A deficiency in Vitamin B6 can be absolutely devastating for people suffering with chronic pain.
  • That’s because like magnesium, selenium and zinc, vitamin B6 is crucial for excitatory neurotransmission. 
  • Specifically, it’s required to convert glutamate (your body’s main excitatory neurotransmitter) into GABA (your body’s main inhibitory neurotransmitter) [29]. 
  • So not having enough Vitamin B6 leads to increased levels of excitation (firing of pain signals) and reduced levels of GABA inhibition, thereby facilitating NMDA overdrive and sensitization of your central nervous system. [30]

Vitamin B6 should also be part of the discussion for anyone dealing with arthritis or neuropathy. Low levels of B6 can be seen in patients suffering from peripheral neuropathy of various etiologies.

There are strong and consistent associations between vitamin B6 status and several indicators of inflammation in patients with rheumatoid arthritis. 

  • Patients with rheumatoid arthritis had low plasma pyridoxal 5′-phosphate compared with healthy control subjects [31]
  • Numerous studies have shown subjective improvements of neuropathy symptoms in patients suffering from Peripheral Neuropathy of various etiologies following   B6 supplementation. [31]

Bonus:

Vitamin B6 has also been shown to improve sleep, lower pro-inflammatory cytokines like IL-6 and TNF-α, reduce pain and disease severity in conditions like fibromyalgia. [32,33]

L-Theanine

The Soothing Amino Acid: regulates glutamate signaling

L-theonine is an amino acid that’s predominantly found in green and black tea.
  • And even though L-Theonine doesn’t act of your NMDA receptors directly, it indirectly stabilizes the excitatory/inhibitory balance inside your brain by increasing GABA – your brain’s natural tranquilizer – while simultaneously calming glutamate. [34]
  • By reducing stress-induced glutamatergic surges, L-theanine can help dampen pain perception and support resilience in those with central sensitization.
  • Helps reduce stress, anxiety, and pain amplification from emotional or mental triggers.

“The green tea amino acid, L-theanine (L-THE), has been shown to induce relaxation, reduce stress, anxiety, and depressive symptoms by influencing the several neurotransmitters associated with the sleep–wake cycle.” [39]

L-theanine can help transform the mental state of people struggling with chronic pain. It promotes alpha brain waves in the brain which is associated with a calm, alert mental state and improved emotional regulation. [35,36]

And like Vitamin B6 L-Theonine also has powerful neuroprotective effects. It Reduces oxidative stress and neuroinflammation, protecting against nerve damage. [37]

Ideal for nervous system regulation and building resilience against the stresses of daily life.

Bonus:

L-Theonine also helps people get to sleep faster while simultaneously improving sleep quality and duration [38,39]
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Synergistic Benefit:

By combining Magnesium + Zinc + Selenium, Vitamin B6 and L-Theanine help dial down the NMDA overdrive in your brain and spinal cord—offering a natural, science-backed pathway to real calm and lasting relief.

Synergistic Mechanism:

Together, these five compounds intervene at multiple points in the pain processing pathway—helping to normalize NMDA receptor activity, reduce neuroinflammation, and restore nervous system balance.

The Solution: Restore Neurological Balance and Reduce Pain with Real Calm

REAL CALM offers a targeted, evidence-based approach to support people suffering with chronic pain, fibromyalgia, neuropathy, and stress-exacerbated flare-ups.

REAL CALM is designed to address NMDA overdrive at its root, by calming the hyperactive NMDA receptors involved in pain amplification, stress reactivity, and central sensitization.

REAL CALM delivers targeted nutrients to calm those signals at their source.

Each ingredient was selected based on its proven ability to regulate glutamatergic activity, protect against neuroinflammation, and rebalance the pain threshold—without sedation or dependency.

Dr. Yoni Whitten

Trusted by Experts. Loved by Clients.

“As a clinician and chronic pain specialist, I’ve seen first-hand how powerful NMDA modulation can be. REAL CALM is a natural extension of what I teach in my clinic—it helps the nervous system shift from survival to healing mode.”
— Dr. Yoni Whitten, Founder of The Pain Fix Protocol™

Who is REAL CALM For?

Individuals with chronic pain, fibromyalgia, migraines, or tension headaches

People experiencing central sensitization or overactive stress responses

Those seeking a non-sedating, non-addictive approach to nervous system regulation

Anyone struggling with pain flare-ups triggered by stress, poor sleep, or inflammation

Real Ingredients. Real Science. Real Calm.

Thousands of people are stuck in the pain-stress cycle. You don’t have to be one of them.

Take back control of your nervous system—naturally.

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References

  1. Li XH, Miao HH, Zhuo M. NMDA Receptor Dependent Long-term Potentiation in Chronic Pain. Neurochem Res. 2019 Mar;44(3):531-538. doi: 10.1007/s11064-018-2614-8. Epub 2018 Aug 14. PMID: 30109556; PMCID: PMC6420414.
  2. Yang S, Seo H, Wang M, Arnsten AFT. NMDAR Neurotransmission Needed for Persistent Neuronal Firing: Potential Roles in Mental Disorders. Front Psychiatry. 2021 Apr 9;12:654322. doi: 10.3389/fpsyt.2021.654322. PMID: 33897503; PMCID: PMC8064413.
  3. Chen SR, Samoriski G, Pan HL. Antinociceptive effects of chronic administration of uncompetitive NMDA receptor antagonists in a rat model of diabetic neuropathic pain. Neuropharmacology. 2009 Aug;57(2):121-6. doi: 10.1016/j.neuropharm.2009.04.010. Epub 2009 May 5. PMID: 19422840; PMCID: PMC2743295.
  4. Kannampalli P, Sengupta JN. Role of principal ionotropic and metabotropic receptors in visceral pain. J Neurogastroenterol Motil. 2015 Mar 30;21(2):147-58. doi: 10.5056/jnm15026. PMID: 25843070; PMCID: PMC4398235.
  5. Volcheck MM, Graham SM, Fleming KC, Mohabbat AB, Luedtke CA. Central sensitization, chronic pain, and other symptoms: Better understanding, better management. Cleve Clin J Med. 2023 Apr 3;90(4):245-254. doi: 10.3949/ccjm.90a.22019. PMID: 37011956.
  6. Nijs J, George SZ, Clauw DJ, Fernández-de-Las-Peñas C, Kosek E, Ickmans K, Fernández-Carnero J, Polli A, Kapreli E, Huysmans E, Cuesta-Vargas AI, Mani R, Lundberg M, Leysen L, Rice D, Sterling M, Curatolo M. Central sensitisation in chronic pain conditions: latest discoveries and their potential for precision medicine. Lancet Rheumatol. 2021 May;3(5):e383-e392. doi: 10.1016/S2665-9913(21)00032-1. Epub 2021 Mar 30. PMID: 38279393.
  7. Kindler LL, Bennett RM, Jones KD. Central sensitivity syndromes: mounting pathophysiologic evidence to link fibromyalgia with other common chronic pain disorders. Pain Manag Nurs. 2011 Mar;12(1):15-24. doi: 10.1016/j.pmn.2009.10.003. Epub 2009 Dec 2. PMID: 21349445; PMCID: PMC3052797.
  8. Nijs J, Goubert D, Ickmans K. Recognition and Treatment of Central Sensitization in Chronic Pain Patients: Not Limited to Specialized Care. J Orthop Sports Phys Ther. 2016 Dec;46(12):1024-1028. doi: 10.2519/jospt.2016.0612. PMID: 27903159.
  9. Nijs J, Torres-Cueco R, van Wilgen CP, Girbes EL, Struyf F, Roussel N, van Oosterwijck J, Daenen L, Kuppens K, Vanwerweeen L, Hermans L, Beckwee D, Voogt L, Clark J, Moloney N, Meeus M. Applying modern pain neuroscience in clinical practice: criteria for the classification of central sensitization pain. Pain Physician. 2014 Sep-Oct;17(5):447-57. PMID: 25247901.
  10. Nijs J, George SZ, Clauw DJ, Fernández-de-Las-Peñas C, Kosek E, Ickmans K, Fernández-Carnero J, Polli A, Kapreli E, Huysmans E, Cuesta-Vargas AI, Mani R, Lundberg M, Leysen L, Rice D, Sterling M, Curatolo M. Central sensitisation in chronic pain conditions: latest discoveries and their potential for precision medicine. Lancet Rheumatol. 2021 May;3(5):e383-e392. doi: 10.1016/S2665-9913(21)00032-1. Epub 2021 Mar 30. PMID: 38279393.
  11. Richmond, Linda. Ketamine: Miracle Drug or Double-Edged Sword? Psychiatric News. Volume 59, Number 11. 21 October 2024. https://doi.org/10.1176/appi.pn.2024.11.11.13
  12. Department of Justice, Drug Enforcement Administration. (2020, April). Drug fact sheet: Ketamine.
  13. https://www.uspharmacist.com/article/the-emerging-role-of-nmda-antagonists-in-pain-management
  14. Tarleton EK, Kennedy AG, Rose GL, Littenberg B. Relationship between Magnesium Intake and Chronic Pain in U.S. Adults. Nutrients. 2020 Jul 16;12(7):2104. doi: 10.3390/nu12072104. PMID: 32708577; PMCID: PMC7400867.
  15. Costello RB, Elin RJ, Rosanoff A, Wallace TC, Guerrero-Romero F, Hruby A, Lutsey PL, Nielsen FH, Rodriguez-Moran M, Song Y, Van Horn LV. Perspective: The Case for an Evidence-Based Reference Interval for Serum Magnesium: The Time Has Come. Adv Nutr. 2016 Nov 15;7(6):977-993. doi: 10.3945/an.116.012765. PMID: 28140318; PMCID: PMC5105038.
  16. Begon S, Pickering G, Eschalier A, Dubray C. Magnesium and MK-801 have a similar effect in two experimental models of neuropathic pain. Brain Res. 2000 Dec 29;887(2):436-9. doi: 10.1016/s0006-8993(00)03028-6. PMID: 11134637.
  17. Boulis M, Boulis M, Clauw D. Magnesium and Fibromyalgia: A Literature Review. J Prim Care Community Health. 2021 Jan-Dec;12:21501327211038433. doi: 10.1177/21501327211038433. PMID: 34392734; PMCID: PMC8371721.
  18. Dolati S, Rikhtegar R, Mehdizadeh A, Yousefi M. The Role of Magnesium in Pathophysiology and Migraine Treatment. Biol Trace Elem Res. 2020 Aug;196(2):375-383. doi: 10.1007/s12011-019-01931-z. Epub 2019 Nov 5. PMID: 31691193.
  19. Choi GJ, Kim YI, Koo YH, Oh HC, Kang H. Perioperative Magnesium for Postoperative Analgesia: An Umbrella Review of Systematic Reviews and Updated Meta-Analysis of Randomized Controlled Trials. J Pers Med. 2021 Dec 2;11(12):1273. doi: 10.3390/jpm11121273. PMID: 34945745; PMCID: PMC8708823.
  20. Krall RF, Moutal A, Phillips MB, Asraf H, Johnson JW, Khanna R, Hershfinkel M, Aizenman E, Tzounopoulos T. Synaptic zinc inhibition of NMDA receptors depends on the association of GluN2A with the zinc transporter ZnT1. Sci Adv. 2020 Jul 3;6(27):eabb1515. doi: 10.1126/sciadv.abb1515. PMID: 32937457; PMCID: PMC7458442.
  21. Hotz C., Brown K.H. Identifying populations at risk of zinc deficiency: the use of supplementation trials. Nutr. Rev. 2001;59(3 Pt 1):80–84. doi: 10.1111/j.1753-4887.2001.tb06992.x.
  22. Nazıroğlu M, Öz A, Yıldızhan K. Selenium and Neurological Diseases: Focus on Peripheral Pain and TRP Channels. Curr Neuropharmacol. 2020;18(6):501-517. doi: 10.2174/1570159X18666200106152631. PMID: 31903884; PMCID: PMC7457405.
  23. Chariot P, Bignani O. Skeletal muscle disorders associated with selenium deficiency in humans. Muscle Nerve. 2003 Jun;27(6):662-8. doi: 10.1002/mus.10304. PMID: 12766976.
  24. Navarro-Alarcon M., Cabrera-Vique C. Selenium in food and the human body: a review. Sci. Total Environ. 2008;400(1-3):115–141. doi: 10.1016/j.scitotenv.2008.06.024.
  25. Chen PC, Guo CH, Tseng CJ, Wang KC, Liu PJ. Blood trace minerals concentrations and oxidative stress in patients with obstructive sleep apnea. J Nutr Health Aging. 2013;17(8):639-44. doi: 10.1007/s12603-013-0023-x. PMID: 24097016.
  26. Amadasi A, Bertoldi M, Contestabile R, Bettati S, Cellini B, di Salvo ML, Borri-Voltattorni C, Bossa F, Mozzarelli A. Pyridoxal 5′-phosphate enzymes as targets for therapeutic agents. Curr Med Chem. 2007;14(12):1291-324. doi: 10.2174/092986707780597899. PMID: 17504214.
  27. Muhamad R, Akrivaki A, Papagiannopoulou G, Zavridis P, Zis P. The Role of Vitamin B6 in Peripheral Neuropathy: A Systematic Review. Nutrients. 2023 Jun 21;15(13):2823. doi: 10.3390/nu15132823. PMID: 37447150; PMCID: PMC10343656.
  28. Chiang EP, Smith DE, Selhub J, Dallal G, Wang YC, Roubenoff R. Inflammation causes tissue-specific depletion of vitamin B6. Arthritis Res Ther. 2005;7(6):R1254-62. doi: 10.1186/ar1821. Epub 2005 Sep 13. PMID: 16277678; PMCID: PMC1297572.
  29. Adventure-Heart DJ, Madden NA, Delfabbro P. Effects of Vitamin B6 (Pyridoxine) and a B Complex Preparation on Dreaming and Sleep. Percept Mot Skills. 2018 Jun;125(3):451-462. doi: 10.1177/0031512518770326. Epub 2018 Apr 17. PMID: 29665762.
  30. Huang SC, Wei JC, Wu DJ, Huang YC. Vitamin B(6) supplementation improves pro-inflammatory responses in patients with rheumatoid arthritis. Eur J Clin Nutr. 2010 Sep;64(9):1007-13. doi: 10.1038/ejcn.2010.107. Epub 2010 Jun 23. PMID: 20571496.
  31. Dashwood R, Visioli F. l-theanine: From tea leaf to trending supplement – does the science match the hype for brain health and relaxation? Nutr Res. 2025 Feb;134:39-48. doi: 10.1016/j.nutres.2024.12.008. Epub 2025 Jan 2. PMID: 39854799.
  32. Hidese S, Ogawa S, Ota M, Ishida I, Yasukawa Z, Ozeki M, Kunugi H. Effects of L-Theanine Administration on Stress-Related Symptoms and Cognitive Functions in Healthy Adults: A Randomized Controlled Trial. Nutrients. 2019 Oct 3;11(10):2362. doi: 10.3390/nu11102362. PMID: 31623400; PMCID: PMC6836118.
  33. Williams JL, Everett JM, D’Cunha NM, Sergi D, Georgousopoulou EN, Keegan RJ, McKune AJ, Mellor DD, Anstice N, Naumovski N. The Effects of Green Tea Amino Acid L-Theanine Consumption on the Ability to Manage Stress and Anxiety Levels: a Systematic Review. Plant Foods Hum Nutr. 2020 Mar;75(1):12-23. doi: 10.1007/s11130-019-00771-5. PMID: 31758301.
  34. Chen SM, Wang MH, Soung HS, Tseng HC, Fang CH, Lin YW, Yang CC, Tsai CC. Neuroprotective effect of l-theanine in a rat model of chronic constriction injury of sciatic nerve-induced neuropathic pain. J Formos Med Assoc. 2022 Apr;121(4):802-814. doi: 10.1016/j.jfma.2021.08.023. Epub 2021 Sep 13. PMID: 34531102.
  35. Kim S, Jo K, Hong KB, Han SH, Suh HJ. GABA and l-theanine mixture decreases sleep latency and improves NREM sleep. Pharm Biol. 2019 Dec;57(1):65-73. doi: 10.1080/13880209.2018.1557698. PMID: 30707852; PMCID: PMC6366437.
  36. Bulman A, D’Cunha N, Marx W, Turner M, McKune A, Naumovski N. The Effects of L-Theanine Supplementation on Quality of Sleep: A Systematic Review. Proceedings. 2023; 91(1):32. https://doi.org/10.3390/proceedings2023091032
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